Introduction
Tissue processing Evaluation
Teased fiber preparation Anatomy
Objectives: Pathologic study of nerve tissue is especially
useful for the recognition of interstitial events such as inflammation or
vascular alterations that cannot be inferred from clinical or electrophysiologic
findings and may provide insight into an underlying mechanism or cause.
Three
major types of pathology
Neuronopathy- pathologic changes in the neurons that contribute
the axon
Axonopathy- pathologic changes of the axon
Primary demyelinating
neuropathy- primary demyelinating disorders not related to
axonopathy
The
fresh specimen should be separated into two half, one-half fix in formalin and
the other half fix in gluteraldehyde- formaldehyde. However, everything can be
done with formalin fixed tissue.
The
segment for paraffin sectioning should be infiltrated
in toto, sectioned after infiltration. Excessive loose connective tissue
attached to the nerve bundle should be removed and embedded in a separate block.
In this way, it will be easier to obtain good transverse section of the nerve
bundle.
Good
transverse paraffin sections should be obtained. Initially, HE, trichrome, and
Congo red stained sections plus 5 unstained (immunostaining ready) sections
should be obtained. Elastic stain, PTAH stain, and extra levels should be
ordered if the suspicion of vasculitis is high. (PTAH in fact works better than
trichrome if only the assessment of fiber lost is the only reason for trichrome
stain. In addition, it has the advantage of detecting minute amount of fibrin
deposition).
Semithin sections stained with toulidine blue or acid-fushin/
toulidine blue should be obtained from both ends of the gluteraldehyde fixed
segment.
The rest of the specimen should be submitted for
teased fiber preparation in toto.
Paraffin sections:
Interstitial Inflammation
Vasculitis (± fibrinoid necrosis)
Perineural inflammation
Endoneural inflammation
Granuloma and giant cells
Infarction
Amyloid and other deposition
Perineural fibrosis
Epineural fibrosis
Semithin
Sections
Loss of large myelinated fiber
Loss of small myelinated fiber
Thinnly myelinated fiber
Hypertrophic changes
Fiber Grouping
Endoneural macrophages
Giant axons
Normal
fibers
Wallerian degeneration: these fibers demonstrate fragmented myelin that
forms ovoids and myelin balls over several internodes.
Paranodal swelling: defined as a paranodal expansion of the axonal
diameter exceeding 150% of the average internodal diaeter. It may be present on
one or both sides of the nodal gap.
Paranodal
demyelination: a nodal gap distance, devoid of any myelin, that
exceeds the internodal mean diameter of the same fiber.
Excessive myelin wrinkling: irregular infoldings and superimposed myelin
folds, involving one third or more of the internodal length in such a way that
the greatest diameter is equal toor exceeds 150% of the smallest diameter. Very
difficult to be separated from artifacts.
Intercalated internode: a fiber shows one remyelinated internode
surrounded on both sides by normally myelinated internodes. This is regarded as
a product of the repair process that follows paradnodal demyelination.
Segmental demyelination: active demyelination with myelin fragmentation and
ovoids within one internode that is surrounded by normally myelinated internodes
is an extremely rare change.
Segmental remyelination: a fiber showing two or more remyelinated
intercalated internodes surrounded by normally myelinated internodes.
Regeneration: a sequence of short, thinly myelinated internodes
less than 50% of a normal internode length of the same fiber diameter.
Blood-nerve-barrier: this barrier is formed by the perineurial cells
and the endothelial cells of the blood vessels. This barrier is not present in
the dorsal root ganglia or in autonomic ganglia. These sites in the PNS are
vulnerable to certain toxins such as mercury.
Blood vessels: Occasional thin rims of lymphocytes around small
epineurial vessels can occur in normal nerve or non-inflammatory neuropathies.
Classification
of myelinated fibers according to their caliber:
Group A: largest fibers with fastest conduction (somatic
afferent and efferent). Group A-II, A-II, A-III are afferent and A-alpha,
A-Beta, and A-gamma are efferent.
Group B: preganglionic fibers of the ANS.
Group C: smallest fibers and postganglionic fibers of the
ANS.
Diameter of fibers: Myelinated fibers exhibit a bimodal distribution of
fiber diameter in the normal nerve with peaks of distribution at 5 and 13 micron
and a range of 2 to 20 micron. Most axons above 3 micron are myelinated.
Unmyelinated fibers range from 0.2 to 3 micron and with peaks of distribution at
1.5 micron.
Endoneurium: The endoneurium has positive pressure in reference
to the perineurium. This expansile tendency and the elastic properties of
perineurium create the uniformly circular shape of each fascicle. Except at the
branching point, normal fascicles that are free of artifacts should appear
circular.
Giant fibers: Giant fibers can be seen in toxic neuropathies and
giant axonal neuropathy (an autosomal recessive disorder).
Length of nodes: the distance between each node along a myelinated
fiber is approximately proportional to the thickness of the myelin sheath with a
range of 200 to 1500 micron. The Schwann cell nucleus is usually sited around
the middle of the internode.
Number of fibers: in a transverse section of a human sural nerve,
there are approximately 8,000 myelinated fibers per mm2, whereas the
unmyelinated axons are more numerous at 30,000 per mm2. Unmyelinated fibers are
more numerous than myelinated fibers in mixed peripheral nerves by a factor of 3
or 4:1.
Perineurium: consists of concentric layers of flatten
perineurial cells [EMA(+), S100(-), Leu7(-), Laminin (variable+), collagen 4
(variable+)] separated by layers of collagen. In the sural nerve, there are 8-12
layers of perineurial cells. The number of layers decreases progressively. The
perineurium blends with the pia-arachnoid. EM:
Perineurial cells are joined by tight junction but arachnoidal cells have
intermediate filaments and are interconnected by well-formed desmosomes.
Perineurial cells also have a discontinsuous pericellular basement membrane and
pinocytic vesicles, features that are not seen in arachnoidal cells.
Remak cells: All axonal satellite cells are generally included
under the term "Schwann cells", but some writers distinguish those
that surround unmyelinated fibers as Remak cells.
Renaut bodies: Renaut bodies are present in 2-7.5% of sural nerve
biopsies. They are 30-200 mm oval
to round bodies, usually subperineurially located, with their long axis runing
parallel to the long axis of the nerve. They tend to collapse on formalin fixed
sections but the shape is maintained in gluteraldehyde or B5 fixed tissue. They
may also have a circumferential disposition around the entire fascicle and
produces the so called “subpeirneuraial edema”.On cross sections, they are
whorled fibrillary structures that may contain few vessels and mast cells but
not axons. In glutyaraldehyde or B5 fixed tissue, they are lightly eosinophilic,
and lightly stained with touluidine blue and Alcian blue but not with PAS or
Congo red. They should not be mistaken as amyloid or immunoglobulin deposits.
Type of cells: about 90% of the nuclei in cross sections belong
to Schwann cells, 5% to fibroblasts, and 5% to other cells such as mast cells
and macrophages. Mast cells are rare but are normal constituents of the
endoneurium and are also seen in sensory ganglia and in the epineurial sheath of
peripheral nerve. Macrophages make up about 2-5% of endoneurial cells.
Unmyelinated fiber: they are better visualized by silver technique
than with immunostaining against neurofilament. Schwann cells associated with
unmyelinated fibers are more likely to be GFAP positive.