Rhabdomyolysis, General

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Background     Reference

BACKGROUND AND CLINICAL INFORMATION: Head  

Summary: This can be and often is a life threatening condition. Rhabdomyolysis or necrotizing myopathy refers to dissolution of skeletal muscle leading to the leakage of the sarcoplasmic content. Other than those due to trauma, it can be viewed as an end result of over exhaustion of muscle. It can be caused by a wide variety of etiologies that include alcoholism, malignant hyperthermia, intoxication, genetic and biochemical defects, crush injury and ischemia, and others. In some cases no cause can be found. Myoglobinuria is the salient feature and often the cause of severe clinical consequence particularly renal failure.

 Clinical:  

·         Myoglobinuria: “tea-colored” urine. Determination of myoglobin level in serum and urine is important for diagnosis.

·         CK: The rise in serum myoglobinuria level precedes CK.

·         Muscle: Swollen and tender muscle with limb weakness. The degree of weakness varies widely and must be distinguished from other cases of acute and widespread muscle weakness.

·         Constitutional: Fever, leukocytosis.

·         Acute renal failure: The most important clinical outcome of rhabdomyolysis is acute renal failure due to acute tubular necrosis.

·         Hypokalemia potentiates rhabdomyolysis.

Hypokalemia potentiates rhabdomyolysis.

REFERENCES: Head

Warren JD et al., Muscle Nerve 2002 Mar;25(3):332-47